Pneumoconiosis: Difference between revisions
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==Background== | ==Background== | ||
Pneumoconiosis is a chronic inflammatory lung disease caused by inhalation of particles, typically occupationally related, that typically results in pulmonary fibrosis and eventually restrictive lung disease. | Pneumoconiosis is a chronic inflammatory lung disease caused by inhalation of particles, typically occupationally related, that typically results in pulmonary fibrosis and eventually restrictive lung disease. | ||
===Types based on inhaled particle=== | |||
*Asbestosis: shipping, roofing, plumbing | *Asbestosis: shipping, roofing, plumbing | ||
*Berylliosis: aerospace, fluorescent bulbs | *Berylliosis: aerospace, fluorescent bulbs | ||
| Line 15: | Line 17: | ||
*Features of restrictive and/or obstructive pulmonary dysfunction | *Features of restrictive and/or obstructive pulmonary dysfunction | ||
*If severe, clubbing, cyanosis | *If severe, clubbing, cyanosis | ||
==Differential Diagnosis== | |||
==Evaluation== | ==Evaluation== | ||
| Line 29: | Line 33: | ||
==Management== | ==Management== | ||
*Treatment is supportive | *Treatment is supportive | ||
==Disposition== | |||
==See Also== | |||
==References== | ==References== | ||
*Meyer KC. Beryllium and lung disease. Chest. Sep 1994;106(3):942-6. | *Meyer KC. Beryllium and lung disease. Chest. Sep 1994;106(3):942-6. | ||
*Kreiss K, Miller F, Newman LS, Ojo-Amaize EA, Rossman MD, Saltini C. Chronic beryllium disease--from the workplace to cellular immunology, molecular immunogenetics, and back. Clin Immunol Immunopathol. May 1994;71(2):123-9. | *Kreiss K, Miller F, Newman LS, Ojo-Amaize EA, Rossman MD, Saltini C. Chronic beryllium disease--from the workplace to cellular immunology, molecular immunogenetics, and back. Clin Immunol Immunopathol. May 1994;71(2):123-9. | ||
[[Category:Pulmonary]] | [[Category:Pulmonary]] | ||
Revision as of 11:44, 19 December 2016
Background
Pneumoconiosis is a chronic inflammatory lung disease caused by inhalation of particles, typically occupationally related, that typically results in pulmonary fibrosis and eventually restrictive lung disease.
Types based on inhaled particle
- Asbestosis: shipping, roofing, plumbing
- Berylliosis: aerospace, fluorescent bulbs
- Byssinosis: cotton
- Silicosis: foundries, sandblasting, mines
- Coal worker's lung: coal
- Siderosis: iron
- Stannosis: tin
Clinical Features
- May be asymptomatic, with normal PFTs, especially initially
- Dyspnea
- Crackles on exam
- Features of restrictive and/or obstructive pulmonary dysfunction
- If severe, clubbing, cyanosis
Differential Diagnosis
Evaluation
- Evaluate for and treat for reversible causes of symptoms or exacerbation of symptoms
- CXR:
- Coal workers': diffuse nodular opacities, especially prominent in upper lung
- Silicosis: Large conglomerate densities in upper lung
- Asbestosis: linear streaking at bases, opacities, honeycomb changes if advanced
- CT Chest
- Best imaging for asbestosis, can detect parenchymal fibrosis and characterize pleural plaques
- TB testing
- Increased incidence of TB in patients with silicosis
Management
- Treatment is supportive
Disposition
See Also
References
- Meyer KC. Beryllium and lung disease. Chest. Sep 1994;106(3):942-6.
- Kreiss K, Miller F, Newman LS, Ojo-Amaize EA, Rossman MD, Saltini C. Chronic beryllium disease--from the workplace to cellular immunology, molecular immunogenetics, and back. Clin Immunol Immunopathol. May 1994;71(2):123-9.