Atrial tachycardia: Difference between revisions

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==Background==
==Background==
 
*Also known as focal atrial tachycardia, Paroxysmal Atrial Tachycardia (PAT), unifocal atrial tachycardia, ectopic atrial tachycardia<ref>Patel A, Markowitz SM. Atrial tachycardia: mechanisms and management. Expert Rev Cardiovasc Ther. 2008 Jul;6(6):811-22. PMID 18570619</ref>
*Rate >100 bpm
*Electrical focus that originates outside in the sinus node at a single location
**By comparison, reentrant tachycardias (eg. [[Paroxysmal supraventricular tachycardia|AVRT, AVNRT]]) involve multiple foci/ larger circuits
*Sustained atrial tachycardia can lead to a tachycardia-induced cardiomyopathy.
*Differs from sinus tachycardia in that the impulses are generated by an ectopic focus somewhere within the atrial myocardium rather than the sinus node.
*Multifocal atrial tachycardia is similar, but has P waves with at least three different morphologies.
*The combination of atrial tachycardia plus AV block is particularly common in [[digoxin toxicity]].


==Clinical Features==
==Clinical Features==
 
*Often asymptomatic
*[[Palpitations]]
**Non-specific finding
**Associated with all tachydysrhythmias, not just AT
**Rapid fluttering/throbbing/pounding sensation in the chest or neck
*[[Syncope]]
**Patients with AT rarely present with syncope
**Cerebral hypoperfusion is more common with a ventricular rate >200 bpm
*[[Chest pain]]
**Can present if there is underlying cardiovascular disease
**Represents a worsening of the associated disease
*[[Dyspnea]]
**Can present if there is underlying cardiovascular disease
**Represents a worsening of the associated disease


==Differential Diagnosis==
==Differential Diagnosis==
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==Evaluation==
==Evaluation==
[[File:PMC3665213 CRIM.MEDICINE2013-430862.002.png|thumb|P waves inverted in lead II and AVL while upright in III and AVF, suggesting ectopic atrial tachycardia, most likely originating from the left side.]]
===Workup===
*[[ECG]]


===Diagnosis===
====ECG Features====
*Atrial rate >100 bpm
*P-waves
**Morphology abnormal (when compared with sinus P wave due to ectopic origin)
**Has at least three consecutive identical ectopic p waves
**Axis frequently abnormal (e.g. inverted in inferior leads)
*QRS complexes
**Usually normal morphology (unless pre-existing bundle branch block, accessory pathway, or rate related aberrant conduction)
*Baseline isoelectric (unlike atrial flutter)
*[[AV block]] may be present


==Management==
==Management==
===Unstable===
*[[Cardioversion]]


===Stable===
''Initial treatment may be aimed at rate control, while later resolution of clinical symptoms usually requires restoration of normal sinus rhythm.''
*If [[digoxin toxicity]], stop drug and consider [[Digoxin Immune Fab]]
*[[Beta-blockers]] are first-line, particularly if blood pressure is stable
*Other options include:
**[[Calcium channel blocker]]
**[[Digoxin]]
**[[Amiodarone]]


==Disposition==
==Disposition==


==See Also==
==See Also==
 
*[[Narrow-complex tachycardia]]


==External Links==
==External Links==
 
*[https://litfl.com/atrial-tachycardia-ecg-library/ LITFL - Focal Atrial Tachycardia (FAT)]


==References==
==References==
<references/>
<references/>
[[Category:Cardiology]]
[[Category:Cardiology]]

Latest revision as of 11:00, 22 March 2026

Background

  • Also known as focal atrial tachycardia, Paroxysmal Atrial Tachycardia (PAT), unifocal atrial tachycardia, ectopic atrial tachycardia[1]
  • Rate >100 bpm
  • Electrical focus that originates outside in the sinus node at a single location
    • By comparison, reentrant tachycardias (eg. AVRT, AVNRT) involve multiple foci/ larger circuits
  • Sustained atrial tachycardia can lead to a tachycardia-induced cardiomyopathy.
  • Differs from sinus tachycardia in that the impulses are generated by an ectopic focus somewhere within the atrial myocardium rather than the sinus node.
  • Multifocal atrial tachycardia is similar, but has P waves with at least three different morphologies.
  • The combination of atrial tachycardia plus AV block is particularly common in digoxin toxicity.

Clinical Features

  • Often asymptomatic
  • Palpitations
    • Non-specific finding
    • Associated with all tachydysrhythmias, not just AT
    • Rapid fluttering/throbbing/pounding sensation in the chest or neck
  • Syncope
    • Patients with AT rarely present with syncope
    • Cerebral hypoperfusion is more common with a ventricular rate >200 bpm
  • Chest pain
    • Can present if there is underlying cardiovascular disease
    • Represents a worsening of the associated disease
  • Dyspnea
    • Can present if there is underlying cardiovascular disease
    • Represents a worsening of the associated disease

Differential Diagnosis

Narrow-complex tachycardia

Evaluation

P waves inverted in lead II and AVL while upright in III and AVF, suggesting ectopic atrial tachycardia, most likely originating from the left side.

Workup

Diagnosis

ECG Features

  • Atrial rate >100 bpm
  • P-waves
    • Morphology abnormal (when compared with sinus P wave due to ectopic origin)
    • Has at least three consecutive identical ectopic p waves
    • Axis frequently abnormal (e.g. inverted in inferior leads)
  • QRS complexes
    • Usually normal morphology (unless pre-existing bundle branch block, accessory pathway, or rate related aberrant conduction)
  • Baseline isoelectric (unlike atrial flutter)
  • AV block may be present

Management

Unstable

Stable

Initial treatment may be aimed at rate control, while later resolution of clinical symptoms usually requires restoration of normal sinus rhythm.

Disposition

See Also

External Links

References

  1. Patel A, Markowitz SM. Atrial tachycardia: mechanisms and management. Expert Rev Cardiovasc Ther. 2008 Jul;6(6):811-22. PMID 18570619