Atrial tachycardia: Difference between revisions
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==Background== | ==Background== | ||
*Also known as focal atrial tachycardia, Paroxysmal Atrial Tachycardia (PAT), unifocal atrial tachycardia, ectopic atrial tachycardia | *Also known as focal atrial tachycardia, Paroxysmal Atrial Tachycardia (PAT), unifocal atrial tachycardia, ectopic atrial tachycardia<ref>Patel A, Markowitz SM. Atrial tachycardia: mechanisms and management. Expert Rev Cardiovasc Ther. 2008 Jul;6(6):811-22. PMID 18570619</ref> | ||
*Rate >100 bpm | *Rate >100 bpm | ||
*Electrical focus that originates outside in the sinus node at a single location | *Electrical focus that originates outside in the sinus node at a single location | ||
**By comparison, reentrant tachycardias (eg. AVRT, AVNRT) involve multiple foci/ larger circuits | **By comparison, reentrant tachycardias (eg. [[Paroxysmal supraventricular tachycardia|AVRT, AVNRT]]) involve multiple foci/ larger circuits | ||
*Sustained atrial tachycardia can lead to a tachycardia-induced cardiomyopathy | *Sustained atrial tachycardia can lead to a tachycardia-induced cardiomyopathy. | ||
* | *Differs from sinus tachycardia in that the impulses are generated by an ectopic focus somewhere within the atrial myocardium rather than the sinus node. | ||
* | *Multifocal atrial tachycardia is similar, but has P waves with at least three different morphologies. | ||
*The combination of atrial tachycardia plus AV block is particularly common in [[digoxin toxicity]]. | |||
==Clinical Features== | ==Clinical Features== | ||
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====ECG Features==== | ====ECG Features==== | ||
*Atrial rate >100 bpm | *Atrial rate >100 bpm | ||
*P | *P-waves | ||
* | **Morphology abnormal (when compared with sinus P wave due to ectopic origin) | ||
* | **Has at least three consecutive identical ectopic p waves | ||
* | **Axis frequently abnormal (e.g. inverted in inferior leads) | ||
* | *QRS complexes | ||
**Usually normal morphology (unless pre-existing bundle branch block, accessory pathway, or rate related aberrant conduction) | |||
*Baseline isoelectric (unlike atrial flutter) | |||
*[[AV block]] may be present | *[[AV block]] may be present | ||
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===Stable=== | ===Stable=== | ||
''Initial treatment may be aimed at rate control, while later resolution of clinical symptoms usually requires restoration of normal sinus rhythm.'' | |||
*If [[digoxin toxicity]], stop drug and consider [[ | *If [[digoxin toxicity]], stop drug and consider [[Digoxin Immune Fab]] | ||
*[[Beta | *[[Beta-blockers]] are first-line, particularly if blood pressure is stable | ||
*Other options include: | |||
**[[Calcium channel blocker]] | |||
**[[Digoxin]] | |||
**[[Amiodarone]] | |||
==Disposition== | |||
==See Also== | ==See Also== | ||
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==External Links== | ==External Links== | ||
*https://litfl.com/atrial-tachycardia-ecg-library/ | *[https://litfl.com/atrial-tachycardia-ecg-library/ LITFL - Focal Atrial Tachycardia (FAT)] | ||
==References== | ==References== | ||
<references/> | <references/> | ||
[[Category:Cardiology]] | [[Category:Cardiology]] | ||
Latest revision as of 11:00, 22 March 2026
Background
- Also known as focal atrial tachycardia, Paroxysmal Atrial Tachycardia (PAT), unifocal atrial tachycardia, ectopic atrial tachycardia[1]
- Rate >100 bpm
- Electrical focus that originates outside in the sinus node at a single location
- By comparison, reentrant tachycardias (eg. AVRT, AVNRT) involve multiple foci/ larger circuits
- Sustained atrial tachycardia can lead to a tachycardia-induced cardiomyopathy.
- Differs from sinus tachycardia in that the impulses are generated by an ectopic focus somewhere within the atrial myocardium rather than the sinus node.
- Multifocal atrial tachycardia is similar, but has P waves with at least three different morphologies.
- The combination of atrial tachycardia plus AV block is particularly common in digoxin toxicity.
Clinical Features
- Often asymptomatic
- Palpitations
- Non-specific finding
- Associated with all tachydysrhythmias, not just AT
- Rapid fluttering/throbbing/pounding sensation in the chest or neck
- Syncope
- Patients with AT rarely present with syncope
- Cerebral hypoperfusion is more common with a ventricular rate >200 bpm
- Chest pain
- Can present if there is underlying cardiovascular disease
- Represents a worsening of the associated disease
- Dyspnea
- Can present if there is underlying cardiovascular disease
- Represents a worsening of the associated disease
Differential Diagnosis
Narrow-complex tachycardia
- Regular
- AV Node Independent
- Sinus tachycardia
- Atrial tachycardia (uni-focal or multi-focal)
- Atrial fibrillation
- Atrial flutter
- Idiopathic fascicular left ventricular tachycardia
- AV Node Dependent
- AV Node Independent
- Irregular
- Multifocal atrial tachycardia (MAT)
- Sinus tachycardia with frequent PACs, PJCs, PVCs
- Atrial fibrillation
- Atrial flutter with variable conduction
- Digoxin Toxicity
Evaluation
Workup
Diagnosis
ECG Features
- Atrial rate >100 bpm
- P-waves
- Morphology abnormal (when compared with sinus P wave due to ectopic origin)
- Has at least three consecutive identical ectopic p waves
- Axis frequently abnormal (e.g. inverted in inferior leads)
- QRS complexes
- Usually normal morphology (unless pre-existing bundle branch block, accessory pathway, or rate related aberrant conduction)
- Baseline isoelectric (unlike atrial flutter)
- AV block may be present
Management
Unstable
Stable
Initial treatment may be aimed at rate control, while later resolution of clinical symptoms usually requires restoration of normal sinus rhythm.
- If digoxin toxicity, stop drug and consider Digoxin Immune Fab
- Beta-blockers are first-line, particularly if blood pressure is stable
- Other options include:
Disposition
See Also
External Links
References
- ↑ Patel A, Markowitz SM. Atrial tachycardia: mechanisms and management. Expert Rev Cardiovasc Ther. 2008 Jul;6(6):811-22. PMID 18570619
