Beer potomania syndrome: Difference between revisions

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==Background==
==Background==
The pathophysiology involves the inability to excrete sufficient free water, based on a loss of normal renal urea gradients. Patients may actually be total-body sodium depleted, yet have elevated urinary sodium and fractional sodium excretion due to this disorder of water metabolism. Attention to proper nutrition during the acute illness may obviate the need for potentially hazardous administration of hypertonic saline
A constellation of electrolyte abnormalities that occur secondary to the over consumption of liquid that is electrolyte poor coupled with little other sources of nutrition.  The poor overall electrolyte intake limits the formation of a normal renal urea gradient which cause an inability to excrete sufficient free water. Patients may actually be total-body sodium depleted, yet have elevated urinary sodium and fractional sodium excretion due to this disorder of water metabolism. Attention to proper nutrition during the acute illness may obviate the need for potentially hazardous administration of hypertonic saline


==Diagnosis==
==Diagnosis==
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==Work-Up==
==Work-Up==
Chem 10  
Chem 10  
Osmolality
ADH


==DDx==
==DDx==
Insert
[[Hyponatremia]]
[[Seizures]]
[[Altered Mental Status]]
[[Weakness]]


==Treatment==
==Treatment==
Insert
If seizing or other severe symptoms, use hypertonic saline
Otherwise gentle replacement of electrolytes with close attention paid to diet is important


==Disposition==
==Disposition==
Insert
Admit patient with seizures, AMS, Severe hyponatremia


==See Also==
==See Also==
*[[Alcohol Intoxication]]
*[[Alcohol Intoxication]]
*[[Alcohol Withdrawal]]
*[[Alcohol Withdrawal]]
*[[Wernicke-Korsakoff Syndrome]]


==Source==
==Source==

Revision as of 23:34, 16 March 2015

Background

A constellation of electrolyte abnormalities that occur secondary to the over consumption of liquid that is electrolyte poor coupled with little other sources of nutrition. The poor overall electrolyte intake limits the formation of a normal renal urea gradient which cause an inability to excrete sufficient free water. Patients may actually be total-body sodium depleted, yet have elevated urinary sodium and fractional sodium excretion due to this disorder of water metabolism. Attention to proper nutrition during the acute illness may obviate the need for potentially hazardous administration of hypertonic saline

Diagnosis

  1. a history of chronic alcohol ingestion (in a hypotonic form)
  2. protein malnutrition
  3. signs, symptoms and laboratory values consistent with water intoxication, including hyponatraemia, hypochloraemia and, usually, hypokalaemia
  4. no evidence of another cause of hyponatraemia such as steroid use, diuretic use, hyperlipidaemia, etc.

Work-Up

Chem 10 Osmolality ADH

DDx

Hyponatremia Seizures Altered Mental Status Weakness

Treatment

If seizing or other severe symptoms, use hypertonic saline Otherwise gentle replacement of electrolytes with close attention paid to diet is important

Disposition

Admit patient with seizures, AMS, Severe hyponatremia

See Also

Source

Va Med. 1989 Jun;116(6):270-1. Beer potomania syndrome in an alcoholic. Harrow AS.