Beer potomania syndrome: Difference between revisions

m (Rossdonaldson1 moved page Beer Potomania Syndrome to Beer potomania syndrome)
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==Diagnosis==
==Diagnosis==
#a history of chronic alcohol ingestion (in a hypotonic form)
*a history of chronic alcohol ingestion (in a hypotonic form)
#protein malnutrition
*protein malnutrition
#signs, symptoms and laboratory values consistent with water intoxication, including hyponatraemia, hypochloraemia and, usually, hypokalaemia
*signs, symptoms and laboratory values consistent with water intoxication, including hyponatraemia, hypochloraemia and, usually, hypokalaemia
#no evidence of another cause of hyponatraemia such as steroid use, diuretic use, hyperlipidaemia, etc.  
*no evidence of another cause of hyponatraemia such as steroid use, diuretic use, hyperlipidaemia, etc.  


==Work-Up==
==Work-Up==
Chem 10  
*Chem 10  
Osmolality
*Osmolality
ADH
*ADH


==DDx==
==Differential Diagnosis==
[[Hyponatremia]]
*[[Hyponatremia]]
[[Seizures]]
*[[Seizures]]
[[Altered Mental Status]]
*[[Altered Mental Status]]
[[Weakness]]
*[[Weakness]]


==Treatment==
==Treatment==
If seizing or other severe symptoms, use hypertonic saline
*If seizing or other severe symptoms, use hypertonic saline
Otherwise gentle replacement of electrolytes with close attention paid to diet is important
*Otherwise gentle replacement of electrolytes with close attention paid to diet is important


==Disposition==
==Disposition==
Admit patient with seizures, AMS, Severe hyponatremia
Admit patient with:
*[[Seizures]]
*[[AMS]]
*Severe [[hyponatremia]]


==See Also==
==See Also==
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==Source==
==Source==
Va Med. 1989 Jun;116(6):270-1.  Beer potomania syndrome in an alcoholic.  Harrow AS.
*Va Med. 1989 Jun;116(6):270-1.  Beer potomania syndrome in an alcoholic.  Harrow AS.


[[Category:FEN]]
[[Category:FEN]]
[[Category:Tox]]
[[Category:Tox]]

Revision as of 04:09, 17 March 2015

Background

A constellation of electrolyte abnormalities that occur secondary to the over consumption of liquid that is electrolyte poor coupled with little other sources of nutrition. The poor overall electrolyte intake limits the formation of a normal renal urea gradient which cause an inability to excrete sufficient free water. Patients may actually be total-body sodium depleted, yet have elevated urinary sodium and fractional sodium excretion due to this disorder of water metabolism. Attention to proper nutrition during the acute illness may obviate the need for potentially hazardous administration of hypertonic saline

Diagnosis

  • a history of chronic alcohol ingestion (in a hypotonic form)
  • protein malnutrition
  • signs, symptoms and laboratory values consistent with water intoxication, including hyponatraemia, hypochloraemia and, usually, hypokalaemia
  • no evidence of another cause of hyponatraemia such as steroid use, diuretic use, hyperlipidaemia, etc.

Work-Up

  • Chem 10
  • Osmolality
  • ADH

Differential Diagnosis

Treatment

  • If seizing or other severe symptoms, use hypertonic saline
  • Otherwise gentle replacement of electrolytes with close attention paid to diet is important

Disposition

Admit patient with:

See Also

Source

  • Va Med. 1989 Jun;116(6):270-1. Beer potomania syndrome in an alcoholic. Harrow AS.