Template:Anticholinergic Toxicity Treatement: Difference between revisions
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==Treatment== | |||
#GI decon | #GI decon | ||
# | #*[[Activated Charcoal]] may be effective even >1hr after ingestion (decreased GI motility) | ||
#Sedation | #Sedation | ||
# | #*Decreases the risk of [[hyperthermia]], [[rhabdo]], traumatic injuries | ||
# | #*[[Benzos]] are agents of choice especially increase seizure threshold | ||
#Cholinesterase inhibition | #Cholinesterase inhibition | ||
# | #*Indicated for severe agitation or delirium (esp if unresponsive to [[benzos]]) | ||
# | #*Avoid when cardiac conduction abnormalities are present | ||
# | #*Physostigmine | ||
# | #**Dosing: 0.5-2mg IV over 5min | ||
# | #**Onset of action: 15-20min | ||
# | #**Side effects: bradycardia, dysrhythmias, cholinergic excess | ||
# | #**always have atropine at the bedside for bradycardia or cholinergic excess | ||
Revision as of 21:05, 23 May 2015
Treatment
- GI decon
- Activated Charcoal may be effective even >1hr after ingestion (decreased GI motility)
- Sedation
- Decreases the risk of hyperthermia, rhabdo, traumatic injuries
- Benzos are agents of choice especially increase seizure threshold
- Cholinesterase inhibition
- Indicated for severe agitation or delirium (esp if unresponsive to benzos)
- Avoid when cardiac conduction abnormalities are present
- Physostigmine
- Dosing: 0.5-2mg IV over 5min
- Onset of action: 15-20min
- Side effects: bradycardia, dysrhythmias, cholinergic excess
- always have atropine at the bedside for bradycardia or cholinergic excess