ECGs by diagnosis: Difference between revisions

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==Cardiac Ischemia==
==ACS==
===T Wave===
*See [[ACS - ECG]]
*Distribution
**T wave is normally inverted in aVR; sometimes inverted in III, aVF, aVL, V1
*T-wave inversions in V2-V6 are pathologic
*Morphology
**Inverted, symmetric,
**Transient changes suggests ischemia without infarction
**Persistent changes suggests infarction (troponin elevation usually seen)
*Pseudonormalization
**In presence of baseline TWI (within 1 month), reocclusion causes normalization of TWI
**Should be interpreted as evidence of ischemia
 
===Q Wave===
*Q waves do not always indicate infarction (see DDX below)
*Must distinguish normal septal q waves from pathologic Q waves:
**Normal septal q wave: <0.04s, low amplitude
**Abnormal septal q wave: >0.04s in I OR in II, III, AND aVF OR V3, V4, V5, AND V6
*Q-wave equivalents in the precordial leads:
**R-wave diminution or poor R-wave progression
**Reverse R-wave progression (R waves increase then decrease in amplitude)
***Must distinguish from lead misplacement
**Tall R waves in V1, V2 (representing "Q waves" from posterior infarction)
 
====Q Wave (Pathologic) DDX====
*Ischemic Q waves
*LBBB
*LVH
*Chronic lung disease
*Hypertrophic cardiomyopathy
*Dilated cardiomyopathy
 
===ST Segment Depression===
*Assume posterior STEMI or reciprocal changes to STEMI until proven otherwise
*Indicators of ischemia:
**>0.5mm depression from baseline (especially >1mm) in two or more contiguous leads
**Transient depression
**Morphology that is flat or downsloping
 
===ST Segment Elevation===
*Stage 1
**Timing: 30min - hours
**Finding: hyperacute T waves
***>6mm limb leads
***>10mm precordial leads
**Duration: normalizes in days, weeks, or months
*Stage 2
**Timing: minutes - hours
**Finding: ST segment elevation
***≥0.1mV in two or more contiguous leads
**Duration: ST segment resolution occurs over 72hrs; completely resolves w/in 2-3wks
*Stage 3
**Timing: within 1hr; completed within 8-12hr
**Finding: Q waves
**Duration: persist indefinitely in 70% of cases
 
===Reciprocal Changes===
#Anterior STEMI
##Reciprocal ST-segment depression in at least one of leads II, III, aVF
##Occurs in 40-70% of cases
#Inferior STEMI
##Reciprocal ST-segment depression usually present in I, aVL; often in V1-V3
##Occurs in 56% of cases
#Posterior STEMI
##Reciprocal ST-segment depression in V1-V4
###Differentiate from inf STEMI reciprocal depression based on upright T waves, posterior leads showing STEMI
##Often associated w/ ST-segment elevation in II, III, aVF (inferior involvement)
 
==Anatomical Correlation==
#Posterior
##ST depression in V1-V2
##R-S ratio of >1 in V1 or V2
##ST elevation >1 in V7-9
##Often accompanied by inferior ST changes
##Often accompanied by RV MI
#Anterior
##ST depression in II, III, aVF usually due to high lateral ST elevation (I, aVL)
#Inferior
##Look at I, aVL for reciprocal changes (ST depression or TWI)
###If no changes pt probably not having an inf STEMI


==Aneurysm==
==Aneurysm==
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*[[ST Segment Elevation]]
*[[ST Segment Elevation]]
*[[ST Segment Depression]]
*[[ST Segment Depression]]
==Source==
*Electrocardiography in Emergency Medicine. ACEP Textbook.


[[Category:Cards]]
[[Category:Cards]]

Revision as of 22:01, 2 March 2012

ACS

Aneurysm

  1. ST elevation > 2wk

Pericarditis

Electrolyte Disorders

Hyperkalemia

Hypokalemia

Hypercalcemia

Hypocalcemia

CNS

  1. SAH, IC bleed, CVA
    1. Diffuse wide, deep, blunted, inverted T waves
    2. QT prolongation

Pacemakers

  1. Should be in the apex of R ventricle
  2. ECG should mimic LBBB w/ LAD

Pulmonary Embolism

  1. S1Q3T3 (Sp, not Sn)

See Also