Central retinal vein occlusion: Difference between revisions
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{{Acute vision loss noninflamed DDX}} | {{Acute vision loss noninflamed DDX}} | ||
== | ==Management== | ||
#Consult ophtho and neuro | #Consult ophtho and neuro | ||
#Complex treatment involving lowering IOP, topical steroids, cyclocryotherapy, photocoagulation while managing underlying medical diseases | #Complex treatment involving lowering IOP, topical steroids, cyclocryotherapy, photocoagulation while managing underlying medical diseases | ||
Revision as of 14:44, 18 October 2015
Background
- Risk Factors
- DM
- HTN
- Hypercoagulable states
- Vasculitis
- Glaucoma
Clinical Features
- Loss of vision
- Variable, ranging from vague blurring to rapid, painless monocular vision loss
Diagnosis
- Fundoscopy
- Optic disc edema, dilated and tortuous veins, diffuse retinal hemorrhages ("blood-and-thunder fundus")
Differential Diagnosis
Acute Vision Loss (Noninflamed)
- Painful
- Arteritic anterior ischemic optic neuropathy
- Optic neuritis
- Temporal arteritis†
- Painless
- Amaurosis fugax
- Central retinal artery occlusion (CRAO)†
- Central retinal vein occlusion (CRVO)†
- High altitude retinopathy
- Open-angle glaucoma
- Posterior reversible encephalopathy syndrome (PRES)
- Retinal detachment†
- Stroke†
- Vitreous hemorrhage
- Traumatic optic neuropathy (although may have pain from the trauma)
†Emergent Diagnosis
Management
- Consult ophtho and neuro
- Complex treatment involving lowering IOP, topical steroids, cyclocryotherapy, photocoagulation while managing underlying medical diseases
See Also
Acute Vision Loss (Noninflamed)