Template:Amiodarone adverse effects: Difference between revisions
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===[[Amiodarone]] Adverse Effects=== | <noinclude><languages/></noinclude> | ||
*[[Bradycardia]] | <translate> | ||
===[[Special:MyLanguage/Amiodarone|Amiodarone]] Adverse Effects=== <!--T:1--> | |||
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*[[Special:MyLanguage/Bradycardia|Bradycardia]] | |||
*Hypotension with older solvent-based formulation. Uncommon with newer aqueous formulation. | *Hypotension with older solvent-based formulation. Uncommon with newer aqueous formulation. | ||
*[[Prolonged QT]] | *[[Special:MyLanguage/Prolonged QT|Prolonged QT]] | ||
*[[Thyrotoxicosis]]<ref>Rosen's 8th Edition</ref> | *[[Special:MyLanguage/Thyrotoxicosis|Thyrotoxicosis]]<ref>Rosen's 8th Edition</ref> | ||
**Between 5-20% of patients treated with amiodarone have thyrotoxicosis (higher in areas of iodine deficiency) | **Between 5-20% of patients treated with amiodarone have thyrotoxicosis (higher in areas of iodine deficiency) | ||
*Iodine-induced hyperthyroidism | *Iodine-induced hyperthyroidism | ||
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*Drug-induced destructive thyroiditis | *Drug-induced destructive thyroiditis | ||
**More commonly, the cytotoxic effects of amiodarone destroy thyroid cells, resulting in a release of preformed hormone. | **More commonly, the cytotoxic effects of amiodarone destroy thyroid cells, resulting in a release of preformed hormone. | ||
*[[Amiodarone pulmonary toxicity]] | *[[Special:MyLanguage/Amiodarone pulmonary toxicity|Amiodarone pulmonary toxicity]] | ||
*Hyperpigmentation rash[[File:Amiodarone_hyperpigmentation.jpg|thumbnail]] | *Hyperpigmentation rash[[File:Amiodarone_hyperpigmentation.jpg|thumbnail]] | ||
</translate> | |||
Latest revision as of 08:25, 20 January 2026
Amiodarone Adverse Effects
- Bradycardia
- Hypotension with older solvent-based formulation. Uncommon with newer aqueous formulation.
- Prolonged QT
- Thyrotoxicosis[1]
- Between 5-20% of patients treated with amiodarone have thyrotoxicosis (higher in areas of iodine deficiency)
- Iodine-induced hyperthyroidism
- It is thought that the iodine load may unmask hyperthyroidism in patients with multinodular goiter and subclinical Graves’ disease
- Drug-induced destructive thyroiditis
- More commonly, the cytotoxic effects of amiodarone destroy thyroid cells, resulting in a release of preformed hormone.
- Amiodarone pulmonary toxicity
- Hyperpigmentation rash
- ↑ Rosen's 8th Edition