Pericardial effusion and tamponade: Difference between revisions

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==Pathophysiology==
==Pathophysiology==
#see pericarditis
#Incr pericardial P > decr diastolic filling > collapse of RA
#hemodynamic compromise from inc pericardial fluid
==Diagnosis==
##incr pericardial pressures
##decr diastolic filling, venous return, collapse of RA
##inhalation sequesters fluid in pulm vasc, not LV
 
==Signs & Symptoms==
#Rapidity of fluid accumulation determines clinical effects
#Rapidity of fluid accumulation determines clinical effects
#Rapid accumulation: as little as 150cc may decr cardiac output
#Symptoms
#Slow accumulation: as much as 2L may have little effect
##CP, SOB, fatigue
#Pts may c/o CP, dyspnea, fatigue, anorexia, wt loss, MS changes, shock
##CHF-type appearance
#Overall: looks a lot like CHF with JVD, SOB, hepatomegaly, etc...
#Signs
#Narrow pulse pressure
##Narrow pulse pressure
#Friction rub
##Friction rub
#Beck's Triad
##Beck's Triad (30% of pts)
##hypotension, muffled heart sounds, JVD
###Hypotension, muffled heart sounds, JVD
##present in only 30% of pts
##Pulsus paradoxus
##90% will have at least one of the three findings
###>10mmHg change in sys BP on inspiration
#Pulsus paradoxus
##Ultrasound
##>10mmHg change in sys BP on inspiration
###RV collapse, effusion
##decreased intrathoracic P on inspiration causes increased return to R heart --> R septum bows into L given constriction by surrounding pericardial effusion --> decreased LVEDV and concomitant decreased CO
##ECG
##can see in many right heart dz states as well
###Normal or low voltage
##may NOT see in acute trauma
###Electrical alternans
#Ewart's sign (compressive atelectasis causes pulmonary auscultatory changes)


==Work-Up==
==Work-Up==
#Pulsus paradoxus (old school)
#ECG
#EKG
#CXR
##nl or diffuse low QRS
#CBC, chem 10, coags, troponin
##electrical alternans (beat to beat QRS amp vary)
##consider ANA, ESR, RF, PPD
#CXR: CM, obliteration of costophrenic angles
#Pericardial fluid
#TTE -modality of choice: effusion, diffuse hypokinesis, RA and RV collapse
##Send for viral/bact Cx, cell count, cytology
#labs: CBC, chem 10, coags, enzymes
#consider: HIV, ANA, ESR, RF, PPD
#pericardial fluid for viral/bact Cx, cell count, cytology


==Etiology==
==Etiology==
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==Disposition==
==Disposition==
#likely ICU
#Likely ICU
#Cards, CT surg consults
#Cards, CT surg consults


==See Also==
==See Also==
Cards: Pericarditis
[[Pericarditis]]


==Source==
==Source==
Adapted from Donaldson
Tintinalli


[[Category:Cards]]
[[Category:Cards]]

Revision as of 03:53, 20 May 2011

Pathophysiology

  1. Incr pericardial P > decr diastolic filling > collapse of RA

Diagnosis

  1. Rapidity of fluid accumulation determines clinical effects
  2. Symptoms
    1. CP, SOB, fatigue
    2. CHF-type appearance
  3. Signs
    1. Narrow pulse pressure
    2. Friction rub
    3. Beck's Triad (30% of pts)
      1. Hypotension, muffled heart sounds, JVD
    4. Pulsus paradoxus
      1. >10mmHg change in sys BP on inspiration
    5. Ultrasound
      1. RV collapse, effusion
    6. ECG
      1. Normal or low voltage
      2. Electrical alternans

Work-Up

  1. ECG
  2. CXR
  3. CBC, chem 10, coags, troponin
    1. consider ANA, ESR, RF, PPD
  4. Pericardial fluid
    1. Send for viral/bact Cx, cell count, cytology

Etiology

  1. Metastatic malignancy
  2. Pericarditis
  3. Uremia
  4. Hemorrhage (anticoagulant)
  5. Other (SLE, postradiation, myxedema)

DDx

  1. Tension PTX
  2. PE
  3. SVC syndrome
  4. large pleural effusion
  5. Tension pneumocardium
  6. Constrictive pericarditis
  7. Cardiogenic shock

Treatment

  1. IV fluids to incr RV vol
  2. Pressors (temporizing)
  3. AVOID preload reducing meds (e.g. nitrates, diuretics)
  4. Procedures
    1. Pericardiocentesis
    2. Pericardial window (OR)

Disposition

  1. Likely ICU
  2. Cards, CT surg consults

See Also

Pericarditis

Source

Tintinalli