Template:Amiodarone adverse effects: Difference between revisions

(Created page with "*Bradycardia *Hypotension with older solvent-based formulation. Uncommon with newer aqueous formulation. *Prolonged QT *Thyrotoxicosis<ref>Rosen's 8th Edition</ref> **...")
 
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*Bradycardia
*[[Bradycardia]]
*Hypotension with older solvent-based formulation. Uncommon with newer aqueous formulation.
*Hypotension with older solvent-based formulation. Uncommon with newer aqueous formulation.
*[[Prolonged QT]]
*[[Prolonged QT]]

Revision as of 13:01, 5 January 2016

  • Bradycardia
  • Hypotension with older solvent-based formulation. Uncommon with newer aqueous formulation.
  • Prolonged QT
  • Thyrotoxicosis[1]
    • Between 5-20% of patients treated with amiodarone have thyrotoxicosis (higher in areas of iodine deficiency)
  • Iodine-induced hyperthyroidism
    • It is thought that the iodine load may unmask hyperthyroidism in patients with multinodular goiter and subclinical Graves’ disease
  • Drug-induced destructive thyroiditis
    • More commonly, the cytotoxic effects of amiodarone destroy thyroid cells, resulting in a release of preformed hormone.
  • Amiodarone Pulmonary Toxicity
  1. Rosen's 8th Edition