Poison Oak, Ivy, Sumac dermatitis: Difference between revisions
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==Background== | ==Background== | ||
[[File:Poison-ivy-oak-sumac.jpg|thumb|]] | |||
[[File:Poison Ivy in Perrot State Park.jpg|thumb|Poison Ivy]] | [[File:Poison Ivy in Perrot State Park.jpg|thumb|Poison Ivy]] | ||
*Type of [[Contact dermatitis|contact dermatitis]] | *Type of [[Contact dermatitis|contact dermatitis]] | ||
| Line 20: | Line 21: | ||
*2 opposing leaflets single terminal leaf | *2 opposing leaflets single terminal leaf | ||
|} | |} | ||
{{Dermatitis types}} | |||
==Clinical Features== | ==Clinical Features== | ||
[[File:PoisonIvyOakSumac.jpeg|thumbnail|Urushiol induced rash]] | [[File:PoisonIvyOakSumac.jpeg|thumbnail|Urushiol induced rash]] | ||
[[File:Urushiol induced contact dermatitis.jpg|thumb|Urushiol induced contact dermatitis]] | [[File:Urushiol induced contact dermatitis.jpg|thumb|Urushiol induced contact dermatitis]] | ||
[[File:Severe allergic reaction to urushiol (poison oak) 4 days after exposure.jpg|thumb|Contact dermatitis from poison oak exposure (Day 4).]] | |||
*Previous exposure leads to sensitization and earlier symptoms | *Previous exposure leads to sensitization and earlier symptoms | ||
**1-2 days for previous sensitizations and 10-14 days for first contact | **1-2 days for previous sensitizations and 10-14 days for first contact | ||
| Line 34: | Line 38: | ||
==Management== | ==Management== | ||
===Prexposure | ===Prophylaxis=== | ||
*Ivy Shield, Stokogard, Organoclay, IvyBlock | ====Prexposure==== | ||
*Ivy Shield, Stokogard, Organoclay, or IvyBlock | |||
===Postexposure | ====Postexposure==== | ||
*Technu Poison Cleanser- may solubilize bound resin several hours post exposure | *Technu Poison Cleanser- may solubilize bound resin several hours post exposure | ||
*Dr Wests Ivy Detox Cleanser chelates urushiol- decreases itching even if have rash | *Dr Wests Ivy Detox Cleanser chelates urushiol- decreases itching even if have rash | ||
===Symptomatic Treatment=== | ===Symptomatic Treatment=== | ||
* | *Soothing measure options: | ||
* | **Oatmeal baths | ||
* | **Cool, wet compresses | ||
**Ice packs | |||
* | **Topical menthol and phenol (calamine lotion) compounds | ||
* | **Topical astringents under occlusion dressing to dry weeping lesions<ref>Poison ivy dermatitis. Baer RL Cutis. 1990;46(1):34</ref> | ||
* | ***Aluminum acetate (Burow's solution) | ||
* | ***Aluminum sulfate calcium acetate (Domeboro) | ||
**Soap mixture of ethoxylate and sodium lauroyl sarcosinate surfactants (Zanfel)<ref>Davila A, Lucas J, Jacoby J, et al. A new topical agent, Zanfel, ameliorates urushiol-induced Toxicodendron allergic contact dermatitis. Ann Emerg Med. 2003;42:S98</ref> | |||
*Oral [[antihistamines]] | |||
**Occasionally used, primarily for sedating effect (itching in poison ivy dermatitis is not caused by histamine release)<ref>Munday J, Bloomfield R, Goldman M, Robey H, Kitowska GJ, Gwiezdziski Z, Wankiewicz A, Marks R, Protas-Drozd F, Mikaszewska M. Chlorpheniramine is no more effective than placebo in relieving the symptoms of childhood atopic dermatitis with a nocturnal itching and scratching component. Dermatology. 2002;205(1):40.</ref> | |||
*High-potency [[topical corticosteroids]]: e.g. [[clobetasol propionate]] 0.05% cream | |||
**No evidence that alters course of disease once vesicles established<ref>Vernon HJ, Olsen EA. A controlled trial of clobetasol propionate ointment 0.05% in the treatment of experimentally induced Rhus dermatitis. J Am Acad Dermatol. 1990;23(5 Pt 1):829.</ref> | |||
*Systemic [[corticosteroids]] for severe disease | |||
**[[prednisone]] taper over 15-20 days: 60mg x 4d then 50mg x 4d then 40 30 20....Use oral steroid for 3wks until skin turns over | |||
**Standard burst dosing may be too short (with rebound dermatitis common)<ref>Brodell RT, Williams L. Taking the itch out of poison ivy. Are you prescribing the right medication? Postgrad Med. 1999;106(1):69.</ref> | |||
===Not Indicated=== | |||
*Topical [[antihistamines]] | |||
*Anesthetics containing [[benzocaine]] | |||
*Antibiotics containing [[neomycin]] or [[bacitracin]] | |||
==Disposition== | |||
*Almost always outpatient | |||
==See Also== | ==See Also== | ||
*[[Rashes]] | *[[Rashes]] | ||
*[[Contact dermatitis]] | *[[Contact dermatitis]] | ||
==References== | ==References== | ||
<references/> | |||
[[Category:Toxicology]] | [[Category:Toxicology]] | ||
Latest revision as of 20:13, 17 April 2024
Background
- Type of contact dermatitis
- Caused by Urashiols found in the sap of the plant.
| Plant | Distribution | Charateristics |
| Ivy | East of the Rockies |
|
| Oak | West of the Rockies |
|
| Sumac | Southeast swamps |
|
Dermatitis Types
- Atopic dermatitis
- Candida dermatitis
- Cercarial dermatitis
- Contact dermatitis
- Dermatitis herpetiformis
- Diaper dermatitis
- Dyshidrotic dermatitis
- Neonatal seborrhoeic dermatitis
- Nummular dermatitis
- Perianal streptococcal dermatitis
- Perioral dermatitis
- Seborrheic dermatitis
- Stasis dermatitis
Clinical Features
- Previous exposure leads to sensitization and earlier symptoms
- 1-2 days for previous sensitizations and 10-14 days for first contact
Differential Diagnosis
Vesiculobullous rashes
Febrile
- Diffuse distribution
- Varicella (chickenpox)
- Smallpox
- Monkeypox
- Disseminated gonococcal disease
- DIC
- Purpural fulminans
- Localized distribution
Afebrile
- Diffuse distribution
- Bullous pemphigoid
- Drug-Induced bullous disorders
- Pemphigus vulgaris
- Phytophotodermatitis
- Erythema multiforme major
- Bullous impetigo
- Localized distribution
- Contact dermatitis
- Herpes zoster (shingles)
- Dyshidrotic eczema
- Burn
- Dermatitis herpetiformis
- Erythema multiforme minor
- Poison Oak, Ivy, Sumac dermatitis
- Bullosis diabeticorum
- Bullous impetigo
- Folliculitis
Evaluation
- Clinical diagnosis
Management
Prophylaxis
Prexposure
- Ivy Shield, Stokogard, Organoclay, or IvyBlock
Postexposure
- Technu Poison Cleanser- may solubilize bound resin several hours post exposure
- Dr Wests Ivy Detox Cleanser chelates urushiol- decreases itching even if have rash
Symptomatic Treatment
- Soothing measure options:
- Oatmeal baths
- Cool, wet compresses
- Ice packs
- Topical menthol and phenol (calamine lotion) compounds
- Topical astringents under occlusion dressing to dry weeping lesions[1]
- Aluminum acetate (Burow's solution)
- Aluminum sulfate calcium acetate (Domeboro)
- Soap mixture of ethoxylate and sodium lauroyl sarcosinate surfactants (Zanfel)[2]
- Oral antihistamines
- Occasionally used, primarily for sedating effect (itching in poison ivy dermatitis is not caused by histamine release)[3]
- High-potency topical corticosteroids: e.g. clobetasol propionate 0.05% cream
- No evidence that alters course of disease once vesicles established[4]
- Systemic corticosteroids for severe disease
- prednisone taper over 15-20 days: 60mg x 4d then 50mg x 4d then 40 30 20....Use oral steroid for 3wks until skin turns over
- Standard burst dosing may be too short (with rebound dermatitis common)[5]
Not Indicated
- Topical antihistamines
- Anesthetics containing benzocaine
- Antibiotics containing neomycin or bacitracin
Disposition
- Almost always outpatient
See Also
References
- ↑ Poison ivy dermatitis. Baer RL Cutis. 1990;46(1):34
- ↑ Davila A, Lucas J, Jacoby J, et al. A new topical agent, Zanfel, ameliorates urushiol-induced Toxicodendron allergic contact dermatitis. Ann Emerg Med. 2003;42:S98
- ↑ Munday J, Bloomfield R, Goldman M, Robey H, Kitowska GJ, Gwiezdziski Z, Wankiewicz A, Marks R, Protas-Drozd F, Mikaszewska M. Chlorpheniramine is no more effective than placebo in relieving the symptoms of childhood atopic dermatitis with a nocturnal itching and scratching component. Dermatology. 2002;205(1):40.
- ↑ Vernon HJ, Olsen EA. A controlled trial of clobetasol propionate ointment 0.05% in the treatment of experimentally induced Rhus dermatitis. J Am Acad Dermatol. 1990;23(5 Pt 1):829.
- ↑ Brodell RT, Williams L. Taking the itch out of poison ivy. Are you prescribing the right medication? Postgrad Med. 1999;106(1):69.
