Budd-Chiari syndrome: Difference between revisions
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==Background== | ==Background== | ||
[[File:Gray1121.png|thumb|Posterior abdominal wall, after removal of the peritoneum, showing kidneys, suprarenal capsules, and great vessels. (Hepatic veins labeled at center top.)]] | |||
*Rare | *Rare | ||
*Caused by occlusion of hepatic veins, 75% primary (thrombosis), 25% secondary (compression by mass) | *Caused by occlusion of hepatic veins, 75% primary (thrombosis), 25% secondary (compression by mass) | ||
*Fulminant, acute, chronic, or asymptomatic. | *Fulminant, acute, chronic, or asymptomatic. | ||
==Clinical Features== | ==Clinical Features== | ||
*Classic triad: | *Classic triad: | ||
Revision as of 14:54, 21 May 2020
Background
- Rare
- Caused by occlusion of hepatic veins, 75% primary (thrombosis), 25% secondary (compression by mass)
- Fulminant, acute, chronic, or asymptomatic.
Clinical Features
- Classic triad:
Differential Diagnosis
Evaluation
- LFTs, BMP, LDH
- Ultrasound, retrograde angiography
- CT and MRI less sensitive
Management
- Sodium restriction, diuretics, anticoagulants
- Venous shunts or TIPS
- Liver transplant
Disposition
See Also
Portal vein thrombosis (PVT)